Hela diskussionen om myocarditis efter COVID-19 är helt absurd. Det är en påhittad skrämselpropaganda som antivaxxar-patrasket sitter och onanerar åt varje gång de läser att en person avlidit i hjärtrelaterade komplikationer. Det faller under den ihopsvamlade föreställningen att ALLA som avlider gör det i komplikationer orsakade av vaccin och är beredda att ljuga hejvilt för att slippa erkänna att de är lågintelligenta idioter som fått allt om bakfoten.
De sista 100 inläggen verkar ha handlat om att slippa erkänna antivaxxar-patrask-lögnerna om att Cristian Eriksen var vaccinerad, och påståendena om att det var vaccinet som orsakade det hjärtstopp han råkade ut för under matchen. Som många påpekat var han INTE vaccinerad. Däremot testade han positivt för COVID-19 en kort tid innan EM 2020.
Men vad säger forskningen? I den här tyska studien tittade man på patienter mellan April-Juni 2020. Alltså långt INNAN något vaccin administrerats. Där visade det sig att 60% hade en pågående myocardial inflammation. 71% hade förhöjda nivåer av Troponin. Över hälften av personerna hade alltså myocarditis efter COVID-19.
Citat:
Results
Of the 100 included patients, 53 (53%) were male, and the mean (SD) age was 49 (14) years. The median (IQR) time interval between COVID-19 diagnosis and CMR was 71 (64-92) days. Of the 100 patients recently recovered from COVID-19, 67 (67%) recovered at home, while 33 (33%) required hospitalization. At the time of CMR, high-sensitivity troponin T (hsTnT) was detectable (greater than 3 pg/mL) in 71 patients recently recovered from COVID-19 (71%) and significantly elevated (greater than 13.9 pg/mL) in 5 patients (5%). Compared with healthy controls and risk factor–matched controls, patients recently recovered from COVID-19 had lower left ventricular ejection fraction, higher left ventricle volumes, and raised native T1 and T2. A total of 78 patients recently recovered from COVID-19 (78%) had abnormal CMR findings, including raised myocardial native T1 (n = 73), raised myocardial native T2 (n = 60), myocardial late gadolinium enhancement (n = 32), or pericardial enhancement (n = 22). There was a small but significant difference between patients who recovered at home vs in the hospital for native T1 mapping (median [IQR], 1119 [1092-1150] ms vs 1141 [1121-1175] ms; P = .008) and hsTnT (4.2 [3.0-5.9] pg/dL vs 6.3 [3.4-7.9] pg/dL; P = .002) but not for native T2 mapping. None of these measures were correlated with time from COVID-19 diagnosis (native T1: r = 0.07; P = .47; native T2: r = 0.14; P = .15; hsTnT: r = −0.07; P = .50). High-sensitivity troponin T was significantly correlated with native T1 mapping (r = 0.33; P < .001) and native T2 mapping (r = 0.18; P = .01). Endomyocardial biopsy in patients with severe findings revealed active lymphocytic inflammation. Native T1 and T2 were the measures with the best discriminatory ability to detect COVID-19–related myocardial pathology.
Conclusions and Relevance
In this study of a cohort of German patients recently recovered from COVID-19 infection, CMR revealed cardiac involvement in 78 patients (78%) and ongoing myocardial inflammation in 60 patients (60%), independent of preexisting conditions, severity and overall course of the acute illness, and time from the original diagnosis. These findings indicate the need for ongoing investigation of the long-term cardiovascular consequences of COVID-19.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7385689/
En annan studie visade att 37% av smittade personer hade pågående pericarditis, myopericarditis eller myocarditis 10 veckor efter att de diagnostiserats med COVID-19. Studien publicerades Juli 2020 och även denna gången finns inget vaccin inblandat. Notera att majoriteten av patienter i studien är medleålders kvinnor - alltså den grupp som har LÄGST risk att drabbas av myocarditis.
Citat:
Results
Median age was 52 years (IQR 41–57), 100 (72%) were women, and 23 (16%) were previously hospitalized for Covid-19 pneumonia. At examination (10.4 [9.3–11.0] weeks after infection-like symptoms), all participants presented hemodynamic stability. Chest pain, dyspnoea or palpitations were observed in 58 (42%) participants; electrocardiographic abnormalities in 69 (50%); NT-pro-BNP was elevated in 11 (8%); troponin in 1 (1%); and CMR abnormalities in 104 (75%). Isolated pericarditis was diagnosed in 4 (3%) participants, myopericarditis in 15 (11%) and isolated myocarditis in 36 (26%). Participants diagnosed by RT-PCR were more likely to still present symptoms than participants diagnosed by serology (73 [71%] vs 18 [50%]; p=0.027); nonetheless, the prevalence of pericarditis or myocarditis was high in both groups (44 [43%] vs 11 [31%]; p=0.238). Most participants (101 [73%]) showed altered immune cell counts in blood, particularly decreased eosinophil (37 [27%]; p<0.001) and increased CD4-CD8-/loTαβ-cell numbers (24 [17%]; p<0.001). Pericarditis was associated with elevated CD4-CD8-/loTαβ-cell numbers (p=0.011), while participants diagnosed with myopericarditis or myocarditis had lower (p<0.05) plasmacytoid dendritic cell, NK-cell and plasma cell counts and lower anti-SARS-CoV-2-IgG antibody levels (p=0.027).
Conclusions
Pericarditis and myocarditis with clinical stability are frequent long after SARS-CoV-2 infection, even in presently asymptomatic subjects. These observations will probably apply to the general population infected and may indicate that cardiac sequelae might occur late in association with an altered (delayed) innate and adaptative immune response.
https://www.medrxiv.org/content/10.1101/2020.07.12.20151316v1
Det finns alltså flertalet studier som visar att så många som 60% av alla som får COVID-19 drabbas av myocarditis.
Hela problematiken med myocarditis har ju flaggats upp från medicinskt håll långt INNAN en enda person blev vaccinerad. Hur i h-e kan man vara så blåsta som individerna ingående i det globala antivaxxar-patraskeriet? Och dessutom skylta med det genom att ideligen länka till Bitchute, Rumble och nu även Reddit? Det är som att kalla in Runar Sögard som karaktärsvittne i en rättegång om ekonomiskt fiffel.
Det får faktiskt vara slut på detta evinnerligt eländiga och amatörmässiga antivaxxar-patraskeriet.
