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But the researchers explain that those spike protein antibodies may themselves produce a second wave of antibodies, called anti-idiotype antibodies or Ab2s. Those Ab2s may modulate the immune system’s initial response by binding with and destroying the first wave of antibodies.

Unfortunately, to do so, the Ab2s must contain structures very similar to the spike protein itself. If they are too similar, they can wind up binding to the same parts of our cells that the spike protein targets.

As the researchers explain:

Some of the resulting anti-idiotype (or “Ab2”) antibodies that are specific for Ab1 can structurally resemble that of the original antigens themselves. Thus, the Ab2 antigen-binding region can potentially represent an exact mirror image of the initial targeted antigen [NOTE: this means the spike protein] in the Ab1 response…

As a result of this mimicry, Ab2 antibodies also have the potential to bind the same receptor that the original antigen was targeting. Ab2 antibodies binding to the original receptor on normal cells therefore have the potential to mediate profound effects on the cell that could result in pathologic changes, particularly in the long term — long after the original antigen itself has disappeared. [Emphasis added.]

In other words, Ab2 antibodies may continue to damage our bodies long after we have cleared either Sars-Cov-2 itself or the spike proteins that the vaccines cause our cells to make.

To be clear, the researchers did not provide proof that these anti-idiotype antibodies are actually causing problems, or even that they exist. The paper merely presents a theory. But the writers believe it could explain the high incidence of myocarditis “after vaccine administration” and even “neurologic effects of SARS-COV-2 infection or vaccines.”