Citat:
Ursprungligen postat av
MrAsparagus
Ett amnat citat:
"Therese Johaug har aldrig gömt sig för några kontroller, preparatet hon åkte dit för är en steroid som inte går att dölja, det finns en klart rimlig förklaring."
Här är han ute och cyklar igen. Vi har konstaterat att detta preparat var enkelt att dölja fram till 2015.
Är vi säkra på detta? Varför har ingen nämnt det i media än i så fall?
Min uppfattning är att salvan är en cover up. Nästan alla som har åkt fast för doping med clostebol har skyllt på salvan.
Carolina Klüfts tidigare konkurrent i längdhopp, brasilianskan Maurren Maggi, stängdes av i två år 2003.
Clostebol innebär att muskelstyrkan i kroppen ökar och brasilianskan skyllde på den sårläkande krämen efter att ha rakat bikinilinjen.
– Jag ville ta bort hår som bildats, sa Maggi då.
För idrottare som riskerar dopingtest är oralt intag av Clostebol det som är svårast att upptäcka. Observera att fram till i år kan Clostebol ha varit ett av de säkraste anabola preparaten att använda utan att riskera upptäckt.
Detta saxat från en dopingexpert:
by Anthony Roberts
"Clostebol is an old anabolic steroid that wasn’t very popular outside of the former German Democratic Republic. In it’s original incarnation, it was known as Megagrisevit-Mono, and was comprised of the parent hormone clostebol (4-chloro-testosterone) attached to the acetate ester.
The acetate ester is unique in the fact that it can be attached to a parent hormone to either produce a short acting injectable compound (think Trenbolone acetate), as well as an oral version (as we see with Primobolan tablets, aka Methenolone acetate). In terms of mass produced anabolic steroids, Primobolan is the only one (off the top of my head) that I can think of, where an acetate ester is used to make a pill. Injectable Primo was only produced (by legitimate pharmaceutical firms) with the Enanthate (long acting) ester.
In fact, the only other steroid commonly attached to this ester is Trenbolone, which although some underground labs offer in pill form, is most commonly found as an acetate in Finaplex pellets – yes, these are the pellets that ranchers inject into cattle to increase slaughter weight. Although it would seem logical that Finaplex pellets could be eaten, I’ve almost never heard of anyone doing that, with most users choosing to grind them up and homebrew their own short-acting injectable. Conversely, I would assume that most athletes subject to drug testing, and using clostebol, would opt for oral ingestion to reduce clearance time. This is what we typically see with drug-tested athletes using winstrol (stanozolol), which is a steroid that features methylation for oral use, but is also available as an injectable (yes, you can simply drink the injectable form of this steroid).
Therefore, Clostebol (which is just testosterone with a 4-chloro substitution), is considerably unique in the fact that it utilizes the same ester for both injectable and oral use. This also makes it harder to detect as an oral, as it doesn’t produce long-acting methylated metabolites.
Besides the (legitimate) oral and injectable versions of the drug (which again, have been discontinued), it’s been produced in a bunch of wacky forms that I can’t imagine are very popular with athletes: an intra-vaginal pill, a bunch of different cremes, and even an intranasal spray. The intranasal spray might offer some additional protection against drug tests, as it would be rapidly absorbed and cleared.
Still, it’s been a relatively under-researched anabolic steroid, so human data on its actual effects is somewhat limited. In fact, until 1993, there was almost no information on the metabolism of clostebol. despite the fact that anabolic steroids had been banned at the Olympic Games since 1976 (testing began in 1984). In 1993, the first paper examining clostebol’s metabolites was published – although hardly any had been characterized, and anything resembling long-term detection was limited to virtually one major metabolite (4-chloroandrost-4-en-3a-ol-17-one). Until recently, this meant that the detection window was relatively small (despite oft-referenced online “detection times”). However, as of 2016, new technology has increased the detection window to at least 31 days.
Unfortunately, while it doesn’t produce many side-effects, it doesn’t produce an overwhelming anabolic effect either. This is likely the reason that it’s been replaced on the legitimate (and illegitimate) market by other (stronger) compounds
So while clostebol is relatively weak, expensive, and rare, the trade-off is worth it to many, who are looking to avoid side-effects…and perhaps beat a drug test. Currently, it’s available on the black market, with both the acetate ester as well as a more long-acting form, which features the same clostebol hormone attached to the enanthate ester. The former would be expected to have an active life of a few hours if consumed orally, or a couple of days if injected."