Citat:
Ursprungligen postat av
kalleboool
Vänligen läs exempelvis följande länk eller anmäl dig till närmsta komvux.
https://www.styrkelabbet.se/aspartam/
Har du själv läst artikeln och dess källor?
Skippar första källan då den inte talar om aspartam utan enbart diabetes och insulin.
Källa #2.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5045440/ Fowler SPG. Low-calorie sweetener use and energy balance: Results from experimental studies in animals, and large-scale prospective studies in humans. Physiol Behav. 2016 Oct 1;164(Pt B):517–23.
Styrkelabbet hävdar följande:
"Ett motargument är att ämnesomsättningen hos en redan överviktig person kan vara mer benäget att reagera negativt på sötningsmedel, inte minst av aspartam – något som har observerats hos möss med ärftlig benägenhet att utveckla fetma och diabetes (Fowler, 2017)."
"Sötningsmedel som inte ger energi, däribland aspartam, har visat sig ge ett antal förändringar i variabler som är relaterade till kroppsvikten hos försöksdjur (Fowler, 2017). Här hittar vi födointag, viktförändring och fettväv."
Står det i Styrkelabbets artikel.
Tittar vi då i studien så framgår även följande: There now exists a body of evidence, from a number of investigators, that animals chronically exposed to any of a range of LCSs – including saccharin, sucralose, acesulfame potassium, aspartame, or the combination of erythritol + aspartame – have exhibited one or more of the following conditions: increased food consumption, lower post-prandial thermogenesis, increased weight gain, greater percent body fat, decreased GLP-1 release during glucose tolerance testing, and significantly greater fasting glucose, glucose area under the curve during glucose tolerance testing, and hyperinsulinemia, compared with animals exposed to plain water or – in many cases – even to calorically-sweetened foods or liquids.
"In addition, the risk of becoming either overweight or obese, among 1,250 participants who had
been normal-weight at baseline, was 93% higher among those in the fourth quartile of
AS beverage intake, compared with non-users. Among 2,571 participants who were
either normal- or overweight (but not obese) at baseline, the risk of incident obesity was
more than doubled among those with the highest ASB intake at baseline, compared
with AS non-users. This difference occurred despite the fact that ASB users, overall –
among participants for whom data from a 24-hour dietary recall were available (for the
first of two SAHS cohorts) – had consumed significantly fewer kilocalories/day at baseline."
Så trots att personerna får i sig mindre kalorier så blir de fetare än de som åt mer vilket kan vara kopplat till dess blockering av IAP och försämring av ämnesomsättningen. Se:
https://www.sciencedaily.com/releases/2016/11/161122193100.htm
"A dramatic, positive dose-response relationship emerged, however, between diet soda
intake at the beginning of each follow-up interval and long-term change in waist
circumference: those who consumed diet sodas daily or more often experienced
subsequent waist circumference gains that were almost quadruple those of non-users;
intermediate waist gains were observed in less-than-daily users, among whom waist
gains were more than double those in non-users58. Increasing abdominal adiposity with
aging has been associated with increased visceral fat59, which in turn has been
associated with increased inflammation60, cardiometabolic risk61;62, and risk of type 2
diabetes60;63, depression64;65, cognitive impairment66, incidence of coronary heart
disease67;68, and mortality69;70. In fact, Després71 has noted that weight loss itself does
not constitute a sufficient goal for intervention, but rather that reducing waist
circumference and increasing physical activity and fitness may be more relevant goals
for health promotion."
"Several ironies have recurred in these animal and human studies: impact has often
been most severe in animals and humans genetically disposed to obesity and diabetes,
or concurrently on a Western-style diet, with high-fat and/or high-sugar content, or
already overweight or obese. Thus, those most at risk often fared the worst by using
LCS. In their 2013 paper, Swithers et al. sounded this cautionary note: ‘These results
suggest that the most negative consequences of consuming high-intensity sweeteners
may occur in those most likely to use them for weight control, females consuming a
“Westernized” diet and already prone to excess weight gain.’19 Another irony is that
cardiometabolic risk has sometimes escalated in LCS-exposed subjects in the absence
of significantly increased weight gain."
Så de negativa effekterna från aspartam klart överstiger de möjliga positiva effekterna man får av en reducerad vikt.
Styrkelabbets egna källa avslutar sin studie med följande:
"What we know for now is this: results from both animal studies and a number of large scale,
long-term observational studies in humans have reported
significantly increased
weight gain, abdominal adiposity, and cardiometabolic risk among those frequently
exposed to LCSs. Even after controlling for a wide range of key potential confounders,
studies in humans have found
significantly increased incidence of depression, metabolic
syndrome, diabetes, cardiovascular events, cardiovascular mortality, and total mortality
among frequent users of the most-widely-used LCS vehicles – diet sodas and other diet
beverages – and these results are troubling."
Detta är ju enbart den första källan i deras artikel. Verkligen nytt lågvattenmärke, att selektivt välja delar av en studie på detta sätt är ju verkligen skamlöst. Anki Sundin är ju en uppenbar aspartam shill eller en idiot och detta var första studien av sju.
