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Du får kanske ta ett uppehåll i thinner-sniffandet och göra som vi andra: läsa vetenskap och sätta oss in i sakfrågan. Att antivaxxar-patrasket skulle ha en aning om vad de pratar om är dock inget man förväntar sig från det packet.
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Findings
A total of 411 myocarditis or pericarditis, or both, events were observed among 15 148 369 people aged 18–64 years who received 16 912 716 doses of BNT162b2 and 10 631 554 doses of mRNA-1273. Among men aged 18–25 years, the pooled incidence rate was highest after the second dose, at 1·71 (95% CI 1·31 to 2·23) per 100 000 person-days for BNT162b2 and 2·17 (1·55 to 3·04) per 100 000 person-days for mRNA-1273. The pooled IRR in the head-to-head comparison of the two mRNA vaccines was 1·43 (95% CI 0·88 to 2·34), with an excess risk of 27·80 per million doses (–21·88 to 77·48) in mRNA-1273 recipients compared with BNT162b2.
A total of 411 myocarditis or pericarditis, or both, events were observed among 15 148 369 people aged 18–64 years who received 16 912 716 doses of BNT162b2 and 10 631 554 doses of mRNA-1273. Among men aged 18–25 years, the pooled incidence rate was highest after the second dose, at 1·71 (95% CI 1·31 to 2·23) per 100 000 person-days for BNT162b2 and 2·17 (1·55 to 3·04) per 100 000 person-days for mRNA-1273. The pooled IRR in the head-to-head comparison of the two mRNA vaccines was 1·43 (95% CI 0·88 to 2·34), with an excess risk of 27·80 per million doses (–21·88 to 77·48) in mRNA-1273 recipients compared with BNT162b2.
https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(22)00791-7/fulltext
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https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8890406/
Myocarditis is the inflammation of the heart muscle and is usually a consequence of a viral infection [1]. Because this disease can cause the destruction of myocytes, it may result in cardiomyopathy, heart failure, and sudden cardiac death. Cardiovascular complications from coronavirus disease 2019 (COVID-19) are emerging [2], especially during hospitalization, and myocarditis has been identified as a cause of death in some COVID-19 patients [3]. In the current epidemiological situation of a very large number of hospitalized patients, we must consider the long-term effects of myocarditis caused by the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Initial reports based on magnetic resonance imaging studies showed 78% of patients with myocardial abnormalities and 60% with ongoing myocarditis 2 to 3 months after COVID-19 infection. High blood troponin concentrations were also found in 76% of patients, although their heart function was preserved [4]. In other studies, about 10 weeks after SARS-CoV-2 infection, 37% of patients were also diagnosed with myocarditis, despite only half of the respondents having symptoms of COVID-19 infection [5]. In contrast, the most recent reports that analyzed data for a fifth of the United States (US) population showed that males between 12 and 17 years of age most likely developed myocarditis within 3 months of SARS-CoV-2 infection, with an incidence of approximately 450 per million infections. The most recent CDC reports, indicating the number of infected teenagers in the US is the highest in all age groups, suggest that myocarditis will become a significant burden [6]. The reports also estimate a 16 times higher risk in patients with COVID-19 compared to the general population, with an incidence of COVID-19-associated myocarditis of approximately 150 cases per 100,000 [7].
Återgå nu till ditt thinner-sniffande och överlåt sakfrågan till oss som har vetenskapliga argument.
