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Ursprungligen postat av
RedX
Angående evolutionära förklaringar:
Människor är som känsligast för sjukdomar när de är väldigt unga (spädbarn). Vilket kön har historiskt ansvarat för spädbarnen?
Spot on my friend.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1810028/pdf/15252963.pdf
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All seven of the disease-salient images were rated as more disgusting by females than males (mean sensitivity: female = 3.5, male = 3.2; two-sample t -test with equal variances; t = 48, p < 0.0001). The difference in score for the disease threat compared with its paired stimulus was also significantly greater for females than males (t = 26.4; p < 0.0001), suggesting that females are responding more sensitively specifically to disease threats than males. Similar results have been found in previous studies (Haidt et al. 1994; Fessler & Navarette 2003). This result is consistent with women’s enhanced evolutionary role in protecting the next generation.
Varför kvinnor känner både mer pathogen disgust och sexual disgust är lätt att förklara. Särskilt skillnaden i sexual disgust är enormt stor, nästan
1,5 standardavvikelser (0,8 standardavvikelser räknas alltså som en stor skillnad). Sexual disgust hos kvinnor ökar under den fertilaste fasen i den menstruella cyckeln, allt för att minimera risken att ligga med fula snubbar med dåliga gener. Variationen i alla subscales av disgust sensitivity (pathogen, sexual, moral) är alltså ärftliga till ungefär 50% och dessutom korrelerade.
https://www.sciencedirect.com/science/article/pii/S1090513803000540
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Disgust operates in many domains of behavior. On the presumption that facets of this emotion evince adaptive design, we conducted a cross-sectional study of 307 women, investigating changes in disgust sensitivity across the menstrual cycle. Two hypotheses were tested, namely (i) sexual disgust is an adaptation that serves to reduce participation in biologically suboptimal sexual behaviors, and (ii) many facets of disgust sensitivity compensate for cyclic changes in immunological robusticity via patterned alterations in behavioral prophylaxis against pathogens. Hypothesis (i) was supported, as disgust sensitivity in the sexual domain, and only in the sexual domain, was positively correlated with presumed conception risk as assessed on the basis of self-reported position in the menstrual cycle. Hypothesis (ii) was not supported, as no facet of disgust sensitivity changed as a function of the presumed level of immunosuppression assessed on the basis of self-reported position in the menstrual cycle. Results are discussed in light of published ethnographic evidence indicating that, in disparate cultures, disgust is elicited by aberrant sexual behaviors, and sex is equated with eating. Together with published findings on an animal model of sexual conditioning, this corpus suggests that sexual disgust may be a panmammalian adaptation.